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Home > JPO > 1995 Vol. 7, Num. 3 > pp. 91-95

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Impression Techniques and Model Modification of a Custom-Molded Ankle-Foot Orthosis for the Idiopathic Clubfoot.

James N. Athearn, CO
Justina S. Case, CO
John M. Roberts, MD

ABSTRACT

With careful impression techniques and model modification methods, a custom ankle-foot orthosis (AFO) may be fabricated to meet the requirements of a physician prescribing noninvasive treatment for postsurgical clubfoot care. The impression is obtained with maximum achievable correction and modified using three-point pressure systems that overcorrect the clubfoot deformity.

This AFO, in the experience of the authors, is effective in protecting the surgical corrections from undue stresses early on as well as helping to prevent postoperative recurrence of the deformity. The postoperative time in a cast also may be shortened if proper orthotic treatment is achieved. Some advantages of a shorter cast period include convenience for patient and family and less risk of skin breakdown.

Introduction

Congenital clubfoot is a deformity noted at birth that includes both idiopathic and nonidiopathic clubfeet (1). The deformity has been found to occur in one to three cases per 1,000 live births (1-3) with a ratio of 2.5 males to 1 female (1).

Although the causes of clubfoot are generally thought to be multifactorial, it has been suggested the uterine environment may interfere with normal embryologic development in these patients (3,4). Other possibilities include a lesion of the peroneal or tibial nerve systems, heredity, bone anomalies and subluxation of the talo-navicular joint (1-4).

More recent information seems to indicate arrested development of the limb in early fetal life results in clubfoot (5-7). The normal fetus passes through a stage of "physiologic clubfoot" during the ninth intrauterine week (8). Factors that indicate a possible arrest in development at this stage include a high rate of associated arterial dysgenesis; the discovery of myofibroblasts, postnatally, on the posterior and medial aspects of the clubfoot; and the diagnosis by ultrasound of the deformity in utero as early as 13 weeks (5-7).

In the idiopathic clubfoot, the foot is the only deformity; the musculoskeletal system is otherwise normal (1,3). The nonidiopathic clubfoot may be present at birth as a local manifestation of other neuromuscular disorders such as arthrogryposis, Larsen's Syndrome, diastrophic dwarfism, myelomeningocele and muscular dystrophy (1-3,9,10). The deformity also may become apparent after birth in pathologies such as cerebral palsy, spina bifida and hydro cephalus (1). The clubfoot deformity seen in association with arthrogryposis, myelomeningocele, muscular dystrophy and other neuropathic diseases tends to be more resistant to treatment even though its cause is understood (1,2). This may be due to muscular imbalances and ligamentous hypertrophy (1,2,4).

A diagnosis of clubfoot can be described as talipes equinovarus. This can be divided into four main deformities:

  • short medial column (adduction and cavus of the foot)
  • hindfoot supination (varus)
  • equinus
  • medial spin (medial transverse rotation of the foot relative to the shin) with lateralization of heel

Treatment for any kind of clubfoot is started as soon as possible after birth (2,4,11). The goal of treatment is to obtain a lasting correction and a foot that is functional and cosmetically accept able (1). Treatment goals also should include making the maximum attempt to maintain normalcy in the lives of the patient and the patient's family (1).

There are many methods of treatment (1,2,4,11,12). The following methods are used at the Shriners Hospital Springfield Unit, where 419 patients with idiopathic clubfoot were under treatment at the time of this study.

Shortly after birth, treatment with serial manipulations and plaster casting is begun. The patient is evaluated after undergoing three months of weekly cast changes with more correction attempted at each cast change.

If a foot seems to correct rapidly, it is probably a nonstructural clubfoot. An ankle-foot orthosis (AFO) is fabricated during the final two weeks of casting and is fit and delivered when the final holding cast is removed. It is worn for 12 out of 24 hours for three to six months with convenience to the family dictating which hours of the day or night it is worn. A Denis-Browne splint for night wear may be indicated to treat persistent medial spin. Follow-up continues on an as-needed basis for at least five to 10 years.

If correction of adduction and supination proceeds rapidly, but the heel remains high and a midfoot breach occurs, a complete posterior release may be indicated. If more than one element of deformity persists after three months of casting, more comprehensive surgery is performed. Postoperative casting continues for at least two months after surgery, followed by application of an AFO.

Postoperatively the foot is placed in an "0" splint, in which it is held in the corrected position with the knee flexed to 90 degrees. The foot and leg are first wrapped with Webril Undercast Padding. Plaster bandage is wrapped around the foot and continued up the medial and lateral sides of the leg and over the knee in a fashion similar to "Robert Jones strapping" as described by Lehman (2). The Webril then is split anteriorly from knee to toes to allow for swelling, and the entire splint is re-wrapped with Kling to hold it in place.

At one week, the splint is removed, the wound is inspected, dressings are applied and a definitive full leg cast is applied. If complete correction is not obtained at this time, serial casting ma be indicated. Postoperative casting continues for two months, and an impression for an AFO is obtained at fly weeks. The AFO is fit and delivered a the time of removal of the final cast Again, if medial spin persists, a Denis Browne splint may be used in conjunction with the AFO.

Methods

One orthosis that is commonly prescribed and fabricated at the Shriners' Hospital as part of both the noninvasive regime and postoperative treatment of the clubfoot is a custom-mold ed AFO with corrective forces based on three-point pressure systems aimed at specific clubfoot deformities (see Figure la and Figure 1b ). The AFO is fabricated from 4-mm (5/32-inch) or 4.8-mm (/6-inch) polypropylene, depending on the size of the child, with a 4.8-mm (3/16inch) Pelite varus prevention pad placed just proximal to the lateral malleolus. It is delivered with Velcro pretibial and ankle straps. The ankle strap may be extended over the dorsum of the foot medially to prevent the great toe from overriding the medial wall. The AFO may be worn at night in conjunction with a Tibial Torsion Transformer or a Denis-Browne bar for treatment of persistent medial spin.

The impression technique for this orthosis is of utmost importance. Maximum correction is desired. Molding with plaster bandage or a synthetic material such as Scotchcast' is acceptable, and landmarks should be outlined.

The impression position should be as follows: If the left foot is being molded, the practitioner's right hand should be holding the calcaneus in a neutral position while pressing the thumb proximal to the lateral malleolus to produce a medially directed force to prevent varus at the ankle. The index finger should create a depression proximal to the insertion of the Achilles tendon to the calcaneus. This will create a pocket in the impression for the heel, which will transfer to the AFO and help prevent the heel from rising into an equinus position relative to the forefoot. The practitioner's left hand should be pushing the forefoot into as much abduction as possible while maintaining the ankle at 90 degrees, and the forefoot should be at a neutral position of pronation/supination.

Model modifications are based on three-point pressure systems for maximum overcorrection. One three-point pressure system consists of a varus prevention pad above the lateral malleolus, the medial border of the foot, and the proximo-medial area of the calf (see Figure 2 ). This three-point pressure system acts on the ankle joint and the subtalar joint to prevent or block varus. The modification of the varus prevention pad is accomplished by removing approximately 3 mm (1/8-inch) of plaster from the model at the position of the thumbprint left from the impression procedure proximal to the lateral malleolus. Wrapping the excavation anteriorly (toward the tibial crest) will result in a varus pad that will reduce the tendency of the AFO to rotate medially around the leg; otherwise medial spin will not be controlled.

A second three-point pressure system (see Figure 3 ) consists of two forces directed laterally-one in the area of the first metatarsal head, the other at the medial side of the calcaneus-and a medially directed counterforce just proximal to the base of the fifth metatarsal and cuboid. This three-point pressure system increases the length of the medial column through the midtarsal, talo-navicular and calcaneo-cuboid joint(s). This is achieved through plaster removal at the medial forefoot of the model. Beginning at the space between the second and third digits, plaster is removed to a point just proximal to the metatarsal heads (essentially removing the first two toes) (see Figure 4a ).This places the forefoot and midfoot in an abducted position in the AFO, which is fabricated with a high medial wall. This overcorrection should be maximal (to the extent tolerable by the patient). The tendency is that the younger the child, the more overcorrection possible. Plaster removal to the third digit is appropriate on an infant. The 5-year-old, on the other hand, may tolerate plaster removal only partially through the first digit.

Approximately 3 mm (1/8-inch) of plaster also must be removed from the model at the medial border proximal to the first metatarsal head to provide relief for the metatarsal head. The plaster removal on the medial side must be compensated for on the lateral side by the addition of an amount of plaster equal to that removed (see Figure 4b ). Plaster buildups also are added over the malleoli for the older child. The infant normally has enough extra tissue that buildups produce only a sloppy fit. Addition of plaster over the heel is avoided to maintain good calcaneal control in the neutral position.

The third three-point pressure system employed maintains the foot at a 90-degree angle to the shin (see Figure 5 ). This three-point pressure system works through the ankle joint to resist equinus. This is the standard force system seen in most AFOs, which directs one upward force at the plantar aspect of the metatarsal heads, another anteriorly directed force at the proximal-posterior calf and a third obliquely directed force at the instep (provided by an ankle strap and/or shoe laces).

The toe plate is extended to encompass the full plantar surface of the foot to ensure the medial wall of the footplate will extend to the end of the toes; this provides the pressure for the overcorrected, abducted position in which the foot is held.

Once the plaster buildups are added and plaster removal is completed, the model is checked to be sure the hindfoot (subtalar joint) is in a neutral inversion/eversion position, the ankle joint is at 90 degrees, and the medial column is lengthened. The model is then smoothed, the trim lines are drawn and the cast is prepared for plastic vacuum-forming with the Pelite varus prevention pad in place over the aforementioned excavation.

Complications

Complications in performing this procedure have included slippage of the AFO distally due to recurrent equinus/heel rise, skin rash and/or breakdown, and shoe-fitting problems.

Slippage has been addressed with the addition of a molded anterior tongue of 1.5-mm (1/16-inch) polyethylene , extending proximally from the top of the varus prevention pad distally to the dorsum of the midfoot. This may be used in conjunction with a 3.2-mm (1/8k inch) Thermofoam pad adhered to the inside of the AFO in the area of the Achilles tendon. Skin rash and breakdown is most often caused by the 23- to 24-hour per day wear of a plastic orthosis. This problem has been alleviated by instructing parents to dress their children in white cotton socks and to change them frequently. A ventilation or "weep" hole may be placed in the posterior-distal area of the calcaneus. To prevent problems of shoe fit, soft Blucher-type sneakers with the innersole(s) removed are recommended.

Conclusion

The authors have found the impression procedures and model modification techniques described herein have been effective in producing an orthosis that maintains the correction of clubfoot deformities that have been effected through manipulation, serial casting and/or surgery. The described AFO has been acceptable to the physician, the patient and the patient's family for preventing recurrence of the clubfoot deformity. The modifications address all aspects of the deformity: shortened medial column, hindfoot supination, equinus and medial spin. The method described herein is recommended as an option for the treatment of clubfoot.


JAMES N ATHEARN, CO, is assistant director of orthotics at Shriners Hospital for Crippled Children, 516 Carew St., Springfield, MA 01104.

JUSTINA S. CASE, CO, was a staff orthotist at Shriners Hospital, Springfield, Mass., during the research and writing of this paper, and is currently employed as a staff orthotist at CEL. Prosthetics and Orthotics, 43 N. Cleveland, Memphis, TN 38104.

JOHN M. ROBERTS, MD, is chief of staff at Shriners Hospital, Springfield, Mass.

References:

  1. Turco V. Current problems in orthopaedics: Clubfoot. New York: Churchill Livingston, 1981:5-6,85-96.
  2. Lehman W. The clubfoot. Philadelphia: Lippincott, 1980:1-6.
  3. Cowell H, Wein B. Current concepts review genetic aspects of club foot. JBJS 1980; 62:1381-7.
  4. Turco V. Present management of the idiopathic clubfoot. J Pedi Orthop, Part B 1994; 3:149-54.
  5. Sodre. Arterial abnormalities in talipes equinovarus as assessed by angiography and the Doppler technique. J Pedi Orthop 1990; 10:101-4.
  6. Zimney JL, Willig SJ, Roberts JM, D'Ambrosia RD. An electronic microscopic study of the fascia from medial and lateral sides of clubfoot. J Pedi Orthop 1985; 5:577-81.
  7. Bronshtein M, Zimmer EZ. Transvaginal ultrasound diagnosis of fetal clubfeet at 13 weeks, menstrual age. J Clin Ultrasound 1989; 17:518-20.
  8. Kawashima T, Uhthoff HK. Development of the foot in prenatal life in relation to the idiopathic clubfoot. J Pedi Orthop 1990; 10:232-7.
  9. Sodergard J, Ryoppy S. Foot deformities in arthrogryposis multiplex congenita. J Pedi Orthop 1994; 14:768-72.
  10. Laville JM, Lakermance P, Limouzy F Larsen's Syndrome: Review of the literature and analysis of 38 cases. J Pedi Orthop 1994; 14:63-73.
  11. McKay DW New concept of and approach to clubfoot treatment: Section II- Correction of the clubfoot. J Pedi Orthop L983; 3:10-21.
  12. Morcuende J, Weinstein 5, Dietz F, Ponseti I. Plaster cast treatment of cluboot: The Ponseti method of manipulation and casting. J Pedi Orthop 1994; 3:161-7.


 

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